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磷酸化IKB α抗体
英文名: Anti-Phospho-NFKBIA (Tyr42)
类别: 其他试剂
021- 联系我时请说是从盖德化工网上看到的
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会员等级： VIP会员 第2年
实名备案： 已实名备案
经济性质： 私营有限责任公司
经营模式： 贸易商,
联系电话： 021-
手机号码： 021-
所在地区： 上海上海
联系地址： 上海市闵行区闵北路88弄1-17号、18-30号第13幢038室
产品详细说明
产品规格：0.2ml/200μg
牌：抗体行业知名品牌
地：美、德及中国大陆
产品包装：管装
格：1元/克
磷酸化IKB &抗体
磷酸化IKB &抗体
&磷酸化IKB &抗体
英文名称& Anti-Phospho-NFKBIA (Tyr42)& &&&&&
中文名称& 磷酸化IKB &抗体 &&&&&
别&&& 名& phospho-NFKBIA(Tyr42); NFKBIA(phospho Y42); IKB-alpha(Phospho-Tyr42); IKB Inhibitor of KB I kappa B I(Kappa)B(alpha); IkappaB IKBA; IKB MAD 3; MAD3; Major histocompatibility complex enhancer binding protein MAD3; NF kappa B NFKBI; NFKBIA; Nuclear factor of kappa light chain gene enhancer in B Nuclear factor of kappa light polypeptide gene enhancer in B cells inhibitor alpha. &&&&&
浓&&& 度& 1mg/1ml &&&&&
规 格& 0.1ml/100&g&&&&&
抗体来源& Rabbit& &&&&&
克隆类型& polyclonal &&&&&
交叉反应& Human, Mouse, Pig, Cow, Rabbit, Sheep &&&&&
产品类型& 一抗& 磷酸化抗体&& &&&&&
研究领域& 肿瘤 免疫学 信号转导 转录调节因子 激酶和磷酸酶&&&&&&&
磷酸化IKB &抗体
蛋白分子量& predicted molecular weight: 35kDa &&&&&
性&&& 状& Lyophilized or Liquid &&&&&
免 疫 原& KLH conjugated Synthesised phosphopeptide derived from human IKB alpha around the phosphorylation site of Tyr42 &&&&&
亚&&& 型& IgG &&&&&
纯化方法& affinity purified by Protein A &&&&&
储 存 液& 0.01M PBS, pH 7.4 with 10 mg/ml BSA and 0.1% Sodium azide &&&&&
产品应用&& WB=1:100-500& ELISA=1:500-1000& IP=1:20-100& IHC-P=1:100-500& IHC-F=1:100-500& IF=1:100-500 &&&&&
（石蜡切片需做抗原修复） &&&&&
¬ yet tested in other applications.&&&&&
&optimal dilutions/concentrations should be determined by the end user.& &&&&&
保存条件& Store at -20 &C for one year. Avoid repeated freeze/thaw cycles. The lyophilized antibody is stable at room temperature for at least one month and for greater than a year when kept at -20&C. When reconstituted in sterile pH 7.4 0.01M PBS or diluent of antibody the antibody is stable for at least two weeks at 2-4 &C. &&&&&
Important Note& This product as supplied is intended for research use only, not for use in human, therapeutic or diagnostic applications. &&&&&
产品介绍 This gene encodes a member of the NF-kappa-B inhibitor family, which contain multiple ankrin repeat domains. The encoded protein interacts with REL dimers to inhibit NF-kappa-B/REL complexes which are involved in inflammatory responses. The encoded protein moves between the cytoplasm and the nucleus via a nuclear localization signal and CRM1-mediated nuclear export. Mutations in this gene have been found in ectodermal dysplasia anhidrotic with T-cell immunodeficiency autosomal dominant disease. [provided by RefSeq, Aug 2011].&&&&&
Function : Inhibits the activity of dimeric NF-kappa-B/REL complexes by trapping REL dimers in the cytoplasm through masking of their nuclear localization signals. On cellular stimulation by immune and proinflammatory responses, becomes phosphorylated promoting ubiquitination and degradation, enabling the dimeric RELA to translocate to the nucleus and activate transcription.&&&&&
Subunit : Interacts with RELA; the interaction requires the nuclear import signal. Interacts with NKIRAS1 and NKIRAS2. Part of a 70-90 kDa complex at least consisting of CHUK, IKBKB, NFKBIA, RELA, IKBKAP and MAP3K14. Interacts with HBV protein X. Interacts with RWDD3; the interaction enhances sumoylation. Interacts (when phosphorylated at the 2 serine residues in the destruction motif D-S-G-X(2,3,4)-S) with BTRC. Associates with the SCF(BTRC) complex, composed of SKP1, CUL1 and BTRC; the association is mediated via interaction with BTRC. Part of a SCF(BTRC)-like complex lacking CUL1, which is associated with RELA; RELA interacts directly with NFKBIA. Interacts with PRMT2. Interacts with PRKACA this interaction is disrupted by thrombin and collagen. Interacts with HIF1AN.&&&&&
Subcellular Location : Cytoplasm. Nucleus. Note=Shuttles between the nucleus and the cytoplasm by a nuclear localization signal (NLS) and a CRM1-dependent nuclear export.&&&&&
Post-translational modifications : P disables inhibition of NF-kappa-B DNA-binding activity. Phosphorylation at positions 32 and 36 is prerequisite to recognition by UBE2D3 leading to polyubiquitination and subsequent degradation.&&&&&
S sumoylation requires the presence of the nuclear import signal. Monoubiquitinated at Lys-21 and/or Lys-22 by UBE2D3. Ubiquitin chain elongation is then performed by CDC34 in cooperation with the SCF(FBXW11) E3 ligase complex, building ubiquitin chains from the UBE2D3-primed NFKBIA-linked ubiquitin. The resulting polyubiquitination leads to protein degradation. Also ubiquitinated by SCF(BTRC) following stimulus-dependent phosphorylation at Ser-32 and Ser-36.&&&&&
Deubiquitinated by porcine reproductive and respiratory syndrome virus Nsp2 protein, which thereby interferes with NFKBIA degradation and impairs subsequent NF-kappa-B activation.&&&&&
DISEASE : Defects in NFKBIA are the cause of ectodermal dysplasia anhidrotic with T-cell immunodeficiency autosomal dominant (ADEDAID) [MIM:612132]. Ectodermal dysplasia defines a heterogeneous group of disorders due to abnormal development of two or more ectodermal structures. ADEDAID is an ectodermal dysplasia associated with decreased production of pro-inflammatory cytokines and certain interferons, rendering patients susceptible to infection.&&&&&
Similarity : Belongs to the NF-kappa-B inhibitor family.&&&&&
Contains 5 ANK repeats.&&&&&
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磷酸化IKB &抗体
Rat L-Selectin ELISA Kit &YT
磷酸化IKB &抗体
说明简要部分，如果需要
磷酸化IKB &抗体
说明书，请来电索取。
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