doi:10.1038/432036a文献中doi是什么意思思

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DOI: 10.1038
DOI Values for: 10.1038
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&, DOI&, DOI.ORG&, and shortDOI& are trademarks of the International DOI Foundation.Nabel, C.S.; Jia, H.; Ye, Y.; Shen, L.; Goldschmidt, H.L.; Stivers, J.T.; Zhang, Y.; Kohli, R.M. AID/APOBEC deaminases disfavor modified cytosines implicated in DNA demethylation. Nat. Chem. Biol. 1–758, doi:10.1038/nchembio.1042.
被如下文章引用:
AUTHORS: ,,,,
KEYWORDS: 5-hydroxymethylcytosine, 5-methylcytosine, cancer, DNA methylation, epigenetics, ten-eleven translocation, TET
JOURNAL NAME:
Sep 07, 2014
ABSTRACT: Prior to 2009, 5-methylcytosine (5-mC) was thought to be the only biologically significant cytosine modification in mammalian DNA. With the discovery of the TET enzymes, which convert 5-methylcytosine (5-mC) to 5-hydroxymethylcytosine (5-hmC), however, intense interest has emerged in determining the biological function of 5-hmC. Here, we review the techniques used to study 5-hmC and evidence that alterations to 5-hmC physiology play a functional role in the molecular pathogenesis of human cancers.

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